A Drop of Blood Tells the Full Story of Heart Attacks

 

Majid Ali, M.D.

344 Prospect Avenue, Suite 1-C, Hackensack,

New Jersey,  07601

201-966-0027


Caution: Please Follow the Treatment of Your Cardiologist Closely If You Have A-Fib or  Other Cardiac Rhythm Disorders.  Do Not Try to Treat Cardiac Emergencies on Your Own. 


 

The heart attack story begins in a drop of blood years before it shows up in the heart. It begins with excess acidity, free radical activity, or Thickness of  the blood which makes blood cell membranes sticky and blood cells to stick to each other and to the cells lining inside of blood vessels called the endo cells. (endo is short for endothelium).


 

We  hope viewers will study the microscopic pictures at the end of this article, understand their meanings, and then study them again with their children to teach them the dangers of eating excess sugary drinks and foods.

These pictures make it easy to learn and understand how diabetes causes poor circulation, heart attacks, and stroke. —for prevention and reversal—more simply and elegantly than any medical textbook or a doctor. Of course, cells can tell their diabetes story only to those who are willing to listen to them. We cannot understand the “cell-speak” if we keep repeating that diabetes is a sugar problem. Sugar is not a cell nor is diabetes cells. Nor can we communicate with cells if our focus is on excess insulin or insulin resistance.


Blood Cells Tell the Cell Membrane Story

Cell membranes exist to separate internal order of cells from their external disorder. They are responsible for cellular cross-talk. So, the health and strength of cells in all parts of the body depend on the the health and strength of their membranes. There are no lab tests available at this time for directly assessing the structural and functional integrity cell membrane in the pancreas (the source of insulin), liver, heart, brain, kidneys, muscles, and other body organs. The state of blood cell membranes, easily evaluated by direct microscopic examination of the blood, provides useful information, albeit indirect, about cell membranes in other body organs. Blood cells tell the story of cell membranes in all organs in the body. body I elaborate this crucial point by presenting the case of insulin and insulin receptor protein embedded in cell membranes.


At the cell membrane level, coronary heart disease develops when blood plasma (the fluid part of blood) becomes acidified, free-radicalized, and curdles.

The blood cells assessing cell membrane practical results The cell membranes become sticky when they become chemicalized—plasticized, so to speak—and hardened, immobilizing the insulin receptors embedded in the membranes. The insulin receptor is a protein that criss-crosses the cell membrane like a cord. One of the consequences of grease buildup on cell membranes is that insulin receptor becomes turned and twisted, literally and figuratively. In a previous paper, I offered the analogy of a crank and a crank-shaft to explain insulin resistance. I visualize insulin as a crank—a device that transmits rotary motion—and the insulin receptor protein as a crank-shaft embedded in the cell membrane receptor protein as a crank-shaft embedded in the cell membranes.


 

Red Blood Cells of a Healthy Person (upper picture) Figure 1

Early Stress on Red Blood Cells (lower picture) Figure 2


Red Blood Cells in a Micro-clot In Uncontrolled Diabetes (upper Picture) Figure 3

Red Blood Cell Clot Breaking Up (lower Picture) Figure 4


Micro-plaque Formation In Uncontrolled Diabetes (both pictures) Figures 5-6


 

Figure 7 (top) illustrates severely damaged erythrocytes in a 52-year-old man with persistent atrial fibrillation. Close examination shows some zones of congealing surrounding many damaged red blood cells.

Figure 8 (bottom) illustrates a zone of plasma congealing unaccompanied by any cellular elements of the blood (seemingly a “spontaneous” phenomenon) in a diabetic with IHD. In our view, such congealing represents accelerated oxidative stress on plasma.


 

Figure 9 (top) shows some needle-like and amorphous granular microclots in a patient with unstable angina.

Figure 10 (bottom) shows a “dirty” blood smear of a man with severe peripheral vascular disease and extensive bilateral leg ulcerations, showing zones of plasma congealing and lumpiness, platelet clumping, and some other zones of plasma congealing unaccompanied by any blood corpuscular elements, representing diffuse changes of AA oxidopathy.


 

Figure 11 (top) shows a microclot formed by a large aggregate of platelets and congealed plasma in a patient five days after angioplasty.

Figure 12 (bottom) shows another field from the same smear and illustrates how microclots in oxidative coagulopathy grow in size when oxidative stress persists.


 

Figure 13 (top) and figure 14 (bottom) show two microplaques in a patient who had received three unsuccessful angioplasties for advanced IHD. Photomicrographs were taken the day after a major nosebleed. Note the compaction of necrotic debris and blood elements in microplaques as contrasted with loose structure of microclots in figure 11.

 

 


Dr. Ali’s No-Cost Heart Course

Heart Disease | The Ali Academy Community – Majid Ali MD

Dec 25, 2014 – Researchers at Harvard have released the following information – Advanced heart failurepatients die less frequently when their cardiologists are away at cardiology conferences. Below is from Harvard: High-risk patients with certain acute heart conditions are more likely to survive than other similar patients …

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In this series of articles and the companion radio and video series on the subject, I will explain and illustrate how the following four blood cell types tell the full diabetes story.

High Blood Insulin and Blood Sugar Levels Increase Blood Acidity and Free Radical Activity, Which Form Microclots and Crystals in the Circulating Blood.

Microclots in Circulating Blood Part 1 from Majid Ali on Vimeo.


Microclots in Circulating Blood Part 2 from Majid Ali on Vimeo.


Red Blood Cells of a Healthy Person (upper picture) Figure 1

Early Stress on Red Blood Cells (lower picture) Figure 2


Red Blood Cells in a Micro-clot In Uncontrolled Diabetes (upper Picture) Figure 3

Red Blood Cell Clot Breaking Up (lower Picture) Figure 4


Micro-plaque Formation In Uncontrolled Diabetes (both pictures) Figures 5-6


 

Figure 7 (top) illustrates severely damaged erythrocytes in a 52-year-old man with persistent atrial fibrillation. Close examination shows some zones of congealing surrounding many damaged red blood cells.

Figure 8 (bottom) illustrates a zone of plasma congealing unaccompanied by any cellular elements of the blood (seemingly a “spontaneous” phenomenon) in a diabetic with IHD. In our view, such congealing represents accelerated oxidative stress on plasma.


 

Figure 9 (top) shows some needle-like and amorphous granular microclots in a patient with unstable angina.

Figure 10 (bottom) shows a “dirty” blood smear of a man with severe peripheral vascular disease and extensive bilateral leg ulcerations, showing zones of plasma congealing and lumpiness, platelet clumping, and some other zones of plasma congealing unaccompanied by any blood corpuscular elements, representing diffuse changes of AA oxidopathy.


 

Figure 11 (top) shows a microclot formed by a large aggregate of platelets and congealed plasma in a patient five days after angioplasty.

Figure 12 (bottom) shows another field from the same smear and illustrates how microclots in oxidative coagulopathy grow in size when oxidative stress persists.


 

Figure 13 (top) and figure 14 (bottom) show two microplaques in a patient who had received three unsuccessful angioplasties for advanced IHD. Photomicrographs were taken the day after a major nosebleed. Note the compaction of necrotic debris and blood elements in microplaques as contrasted with loose structure of microclots in figure 11.


Please support the staff of this website keep the articles posting and categorized by considering these books and  long-playing extended videos at  $9.95 – $19.95 each.

Diabetes Book
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DIABETES – Reversing Diabetes  Part 1 – Dr. Ali’s Course on Healing
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DIABETES – Reversing Diabetes By Regenerating Pancreas Seminar
DIABETES – Dr. Ali’s Course on Reversing Diabetes – Subtypes of Diabetes Type 2: 2A and 2B
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DIABETES – Reversing Diabetes Part 2 – Dr. Ali’s Course on Healing
DIABETES – Reversing Diabetes By Regenerating Pancreas Seminar
DIABETES – Dr. Ali’s Course on Reversing Diabetes – Subtypes of Diabetes Type 2: 2A and 2B
DIABETES – I Have Diabetes. I Have to Lose It. Seminar 1 – Dr. Ali’s Course on Diabetes
DIABETES – I Have Diabetes. I Have to Lose It. Seminar 2 – Dr. Ali’s Course on Diabetes


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